A molecular mechanism at work during periods of stress may shed light on developing new therapies for post-traumatic stress disorder and other conditions. Researchers at UT Southwestern Medical Center say that mice which are vulnerable to stress experience an increase of nerve activity that requires dopamine. Less vulnerable mice express molecules that prevent the release of dopamine. The challenge will be developing new therapies without injuring the patient, which benefits from dopamine in one part of the brain while being injured by it in another.

- read the report [1] from The Washington Post

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